EBV is one of the most common viruses in the world. A person becomes infected during childhood; Infection occurs, as a rule, through biological fluids such as saliva, which persists throughout life in 90% of the human population.
Scientists noted that the virus, when activated, can destroy chromosomes and cause oncology. Work has already been done on this subject, but the latter has become more important.
The researchers studied the EBNA1 virus protein, which persists in infected cells. The researchers found that EBNA1 binds EBV-like sequences at a fragile site on human chromosome 11. And with the increase in the amount of protein in this chromosome, a break occurs that creates the potential for cancer development. EBNA1 also suppresses the p53 gene, which is responsible for cell division and death, thereby preventing tumor formation.
The scientists then analyzed the results of whole genome sequencing of 2,439 cancer cases across 38 tumor types. It was revealed that in the presence of Epstein-Barr virus, cancerous tumors showed a higher level of 11th chromosome abnormalities compared to the absence of EBV, and this correlation was 100% in the presence of head and neck tumors.
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Source: Ferra
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