dietary restrictions. Scientist Clive Mackay discovered that mice fed much less than normal could live much longer than their relatives who were not rationed. They also stayed younger and healthier for longer. But it’s still unclear whether this works in humans. Experts are now working to develop drugs that mimic dietary restriction, such as rapamycin or metformin. They can mimic the effect of a person eating less, without causing constant hunger pangs.

“Little Aging”. People’s risk of dying doubles every eight years. However, some animals have a condition called mini-aging. This is when the risk of death does not depend on how old they are. So they don’t seem to age.

signs of aging. There is now finally a scientific consensus on what causes aging.

telomerase. Telomeres shorten throughout life, and people with shorter telomeres for their age are more at risk of disease in old age and die earlier than those with longer telomeres. But an enzyme called telomerase can lengthen telomeres.

thymus rejuvenation. This organ is responsible for the production of immune cells. As we get older, we become more susceptible to infections because its function is impaired. But scientists have many ideas on how to fix this situation, from gene therapy to stem cells, hormones and drugs.

Induced pluripotent stem cells. These cells are created by taking normal body cells and combining four different genes, allowing them to turn into any type of cell. The most advanced of these is to transform these cells into new eye cells to replace those lost in age-related macular degeneration. Maybe soon they can also be used for Parkinson’s disease, arthritis, thymus contraction and the need for tooth replacement.

Amish gene. Scientists have found that people with one copy of the mutated SERPINE1 gene have better heart health, are less likely to have diabetes, and live up to 10 years longer than those without the disease.

Epigenetic clock. Tracking changes in epigenetics can provide incredibly accurate predictions of how old you are. This would allow for faster testing of various drugs because scientists would need to change people’s epigenetic age before and after taking the drug.

Periodic reprogramming. This process can make cells biologically younger. Experiments on mice have shown that after this procedure, mice can live longer, their health improves, and damaged optic nerve cells regenerate. It is important to safely translate this idea into human therapy.

Senolytic drugs. These drugs destroy senescent cells. These cells also secrete molecules that accelerate the aging process. Their number increases with age, which contributes to degeneration. The first senolytics have now begun to be tested in humans. If successful, they could be the first true anti-aging drug.

News materials cannot be equated with a doctor’s prescription. Consult an expert before making a decision.

Source: Ferra

Previous article5 Great Prequels to See if You Liked Furiosa: From the Mad Max Saga
Next articleiPad Pro M4 OLED turned out to be a tablet with almost no flawsLaptops and tablets18:00 | May 26, 2024
I am a professional journalist and content creator with extensive experience writing for news websites. I currently work as an author at Gadget Onus, where I specialize in covering hot news topics. My written pieces have been published on some of the biggest media outlets around the world, including The Guardian and BBC News.

LEAVE A REPLY

Please enter your comment!
Please enter your name here