In animal models, “genetically modified” mice lacking TRPC5 showed similar symptoms, including weight gain, anxiety, and lack of maternal care. Specifically, these effects were explained by TRPC5’s effect on oxytocin neurons in the hypothalamus of the brain. Known for its role in emotional bonding, oxytocin has been shown to relieve symptoms when these neurons are reactivated.
The discovery opens up new possibilities for treating obesity and postpartum depression by targeting the oxytocin system. The findings highlight the “biological basis” of these diseases and point to “potential avenues for therapeutic intervention,” paving the way for future clinical trials examining the effectiveness of oxytocin in treating people with these diseases, the scientists wrote.
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Source: Ferra
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