A study published in the journal Nature Cardiovascular Research reveals how saturated fatty acids trigger the production of SEMA3A, a signaling molecule that closes critical windows in the liver’s blood vessels. This closure prevents the transport of fat from the liver, exacerbating fat accumulation and potentially leading to liver inflammation and serious complications.
The team showed that inhibiting SEMA3A in mouse models reversed this process, reopening the windows and reducing fat accumulation in the liver, thus improving liver function. The author of the study is Dr. Eckhard Lammert hopes that these findings could pave the way for new therapeutic strategies to prevent and treat FLD, potentially preventing its progression to more serious liver diseases such as cirrhosis and cancer.
Further studies are needed to confirm these results and investigate their applicability in clinical settings.
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Source: Ferra
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